Judith Miklossy , the author of the abstract below, has found in several studies, that different types of spirochetes ( oral spirochetes, Syphilis spirochetes and Lyme spirochetes ), are found in many autopsies of Alzheimer's brains and none in the control groups. Alan Macdonald , another Lyme disease researcher has found the same results. Why are we not hearing about this in the media? The media has predicted that Alzheimer's disease will significantly increase in the coming years. How do they know this? Government health agencies state that this will be due to the increase in the aging population. Healthy aging citizens should NOT get Alzheimer's disease and other forms of dementia do not have to be a normal part of aging. Diet and infections...and perhaps vaccines, play a critical role. The government health agencies/drug companies/media completely avoid discussing the Alzheimer's studies done by the above researchers. Alzheimer's seems to be increasing with the growing number of Lyme disease cases, invasive dental procedures....and the increase in flu shots for the elderly. Some researchers feel that gingivitis and dental work may play a role by providing a direct access to the brain by spirochetes and other microbes... through breeches in the oral cavity. This is known to happen with the heart...heart problems resulting from dental work or poor oral health in general.
The abstract below also speaks about the amyloid plaques in Alzheimer's Disease and how brain colonies of spirochetes can form the filamentous plaques found in Alzheimer's. Let's start talking about this! Alzheimer's may or already has affected someone you know and love. There is a cause so let's not just look at symptomatic treatments....let's find the underlying cause before it is too late!
For more info on bacteria and Alzheimer's Disease....check out the sidebar of this blog.
http://www.miklossy.ch/473.html
http://doi.org/10.3389/fnagi.2015.00046
Free, full text of this Open Access article, (pdf file, 2.5 MB):
http://journal.frontiersin.org/article/10.3389/fnagi.2015.00046/pdf
The abstract below also speaks about the amyloid plaques in Alzheimer's Disease and how brain colonies of spirochetes can form the filamentous plaques found in Alzheimer's. Let's start talking about this! Alzheimer's may or already has affected someone you know and love. There is a cause so let's not just look at symptomatic treatments....let's find the underlying cause before it is too late!
For more info on bacteria and Alzheimer's Disease....check out the sidebar of this blog.
Historic evidence to support a causal
relationship between spirochetal infections and Alzheimer's
disease.
http://doi.org/10.3389/fnagi.2015.00046
Abstract
Following previous observations a statistically significant association between various types of spirochetes and Alzheimer’s disease (AD) fulfilled Hill’s criteria in favor of a causal relationship. If spirochetal infections can indeed cause AD, the pathological and biological hallmarks of AD should also occur in syphilitic dementia.
To answer this question, observations and illustrations on the detection of spirochetes in the atrophic form of general paresis, which is known to be associated with slowly progressive dementia, were reviewed and compared with the characteristic pathology of AD. Historic observations and illustrations published in the first half of the 20th Century indeed confirm that the pathological hallmarks, which define AD, are also present in syphilitic dementia.
Cortical spirochetal colonies are made up by innumerable tightly spiraled Treponema pallidum spirochetes, which are morphologically indistinguishable from senile plaques, using conventional light microscopy. Local brain amyloidosis also occurs in general paresis and, as in AD, corresponds to amyloid beta.
These historic observations enable us to conclude that chronic spirochetal infections can cause dementia and reproduce the defining hallmarks of AD. They represent further evidence in support a causal relationship between various spirochetal infections and AD. They also indicate that local invasion of the brain by these helically shaped bacteria reproduce the filamentous pathology characteristic of AD.
Chronic infection by spirochetes, and co-infection with other bacteria and viruses should be included in our current view on the etiology of AD. Prompt action is needed as AD might be prevented.
Miklossy J.
Frontiers in Aging Neuroscience. 2015 Apr 16;7:46.
eCollection 2015.http://doi.org/10.3389/fnagi.2015.00046
Abstract
Following previous observations a statistically significant association between various types of spirochetes and Alzheimer’s disease (AD) fulfilled Hill’s criteria in favor of a causal relationship. If spirochetal infections can indeed cause AD, the pathological and biological hallmarks of AD should also occur in syphilitic dementia.
To answer this question, observations and illustrations on the detection of spirochetes in the atrophic form of general paresis, which is known to be associated with slowly progressive dementia, were reviewed and compared with the characteristic pathology of AD. Historic observations and illustrations published in the first half of the 20th Century indeed confirm that the pathological hallmarks, which define AD, are also present in syphilitic dementia.
Cortical spirochetal colonies are made up by innumerable tightly spiraled Treponema pallidum spirochetes, which are morphologically indistinguishable from senile plaques, using conventional light microscopy. Local brain amyloidosis also occurs in general paresis and, as in AD, corresponds to amyloid beta.
These historic observations enable us to conclude that chronic spirochetal infections can cause dementia and reproduce the defining hallmarks of AD. They represent further evidence in support a causal relationship between various spirochetal infections and AD. They also indicate that local invasion of the brain by these helically shaped bacteria reproduce the filamentous pathology characteristic of AD.
Chronic infection by spirochetes, and co-infection with other bacteria and viruses should be included in our current view on the etiology of AD. Prompt action is needed as AD might be prevented.
http://www.miklossy.ch/473.html
http://doi.org/10.3389/fnagi.2015.00046
Free, full text of this Open Access article, (pdf file, 2.5 MB):
http://journal.frontiersin.org/article/10.3389/fnagi.2015.00046/pdf
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They have already been many studies in their field where scientists and dentist were able to link dementia and an onset of Alzheimer’s to bad oral care, today this new study has proved it and certainly cemented the facts.
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